Oh wow! <3 Sounds like the kind of thing I’d like to do.
My working hypothesis, thus far, is that an aberrant DA increase in the amygdala in response to extended (or chronic) early life stress exposure (in which CORT levels are increased) may have a role in mediating neurobehavioral dysfunction (or emotional behavior deficits) that emerge later in development.
This is tentative. As I met with my PI today and we’re still discussing our options…
To better sum up my interests, the NIH has a new RO1 that funds research on limbic system/forebrain development and it’s relation to addiction during later life. If I were a PI, this is what I would go for.